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Fatigue Crack Retardation and Retardation Mechanism in Variable Loading (The Effects of Crack Tip Branching in Crack Growth Retardation)
Song. Sam-Hong, Kwon. Yun-Ki 한국정밀공학회 International journal of the Korean society of precision engineering 6 Pages
한국정밀공학회 International journal of the Korean society of precision engineering 2002, Vol.3 No.3 76-81 (6 pages)
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Curcumin protects against the intestinal ischemia-reperfusion injury: involvement of the tight junction protein ZO-1 and TNF-α related mechanism
ShuyingTian, RuixueGuo, SichenWei, YuKong, XinliangWei, WeiweiWang, XiaomengShi, HongyuJiang 대한생리학회-대한약리학회 The Korean Journal of Physiology & Pharmacology 6 Pages
대한생리학회-대한약리학회 The Korean Journal of Physiology & Pharmacology 2016, Vol.20 No.2 2 147-152 (6 pages)
mechanism may also be assumed for I/R injury. Recent study by Shen et al. indicated that I/R insult resulted in the disruption of TJs and an increase in intestinal permeability, Original Article Curcumin protects against the intestinal ischemia-reperfusion injury: involvement of the tight junction protein ZO-- related mechanism Shuying Tian #, Ruixue Guo #, Sichen Wei*, Yu Kong, Xinliang Wei,... Present study aimed to investigate the effect of curcumin-pretreatment on intestinal I/R injury and on intestinal mucosa barrier. Thirty Wistar rats were randomly divided into: sham, I/R, and curcumin groups (n=10). Animals in curcumin group were pretreated with curcumin by gastric gavage (200 mg/kg) for 2 days before I/R. Small intestine tissues were prepared for Haematoxylin & Eosin (H&E) staining. Serum diamine oxidase (DAO) and tumor necrosis factor (TNF)-α levels were measured.... -
Altered Regulation of Renal Acid Base Transporters in Response to Ammonium Chloride Loading in Rats
EunYoungKim, JoonSeokChoi, KoEunLee, ChangSeongKim, EunHuiBae, SeongKwonMa, SuhnHeeKim, JongUnLee, SooWanKim 대한생리학회-대한약리학회 The Korean Journal of Physiology & Pharmacology 5 Pages
대한생리학회-대한약리학회 The Korean Journal of Physiology & Pharmacology 2012, Vol.16 No.2 2 91-95 (5 pages)
in chronic acid loading. These findings suggest that adapted activation of H ＋-ATPase in response to metabolic acidosis might coun- teract the development of metabolic acidosis. In addition to apical membrane H ＋ -ATPase, reabsorb HCO3 － to the blood through basolateral membrane localized AE1 plays a role in acid-base homeostasis. Several lines of evidence indicated that AE1 also provide... a subject of considerable interest for many years. The present study was aimed to determine whether there is an altered regulation of renal acid base transporters in acute and chronic acid loading. Male Sprague-Dawley rats were used. Metabolic acidosis was induced by administration of NH4Cl for 2 days (acute) and for 7days (chronic). The serum and urinary pH and bicarbonate were measured. The protein expression of renal acid base transporters [type 3 Na＋/H＋ exchanger (NHE3), type 1 Na＋/HCO3－... -
Altered Gene Expression in Cerulein-Stimulated Pancreatic Acinar Cells: Pathologic Mechanism of Acute Pancreatitis
JiHoonYu, JooWeonLim, HyeyoungKim 대한생리학회-대한약리학회 The Korean Journal of Physiology & Pharmacology 8 Pages
대한생리학회-대한약리학회 The Korean Journal of Physiology & Pharmacology 2009, Vol.13 No.6 1 409-416 (8 pages)
Seodaemun-gu, Seoul 120-749, Korea. (Tel) 82-2-2123-3125, (Fax) 82-2-364-5781, (E-mail) kim626@yonsei.ac.kr Altered Gene Expression in Cerulein-Stimulated Pancreatic Acinar Cells: Pathologic Mechanism of Acute Pancreatitis Ji Hoon Yu 1 , Joo Weon Lim 2 , and Hyeyoung Kim 2 1Department of Pharmacology, Yonsei University College of Medicine, Seoul 120-752, 2Department of Food and Nutrition,... Acute pancreatitis is a multifactorial disease associated with the premature activation of digestive enzymes. The genes expressed in pancreatic acinar cells determine the severity of the disease. The present study determined the differentially expressed genes in pancreatic acinar cells treated with cerulein as an in vitro model of acute pancreatitis. Pancreatic acinar AR42J cells were stimulated with 10−8 M cerulein for 4 h, and genes with altered expression were identified using a cDNA... -
Mechanism of Acetylcholine-induced Endothelium-dependent Relaxation in the Rabbit Carotid Artery by M3-receptor Activation
Yong-JinSong, Seong-ChunKwon 대한생리학회-대한약리학회 The Korean Journal of Physiology & Pharmacology 6 Pages
대한생리학회-대한약리학회 The Korean Journal of Physiology & Pharmacology 2004, Vol.8 No.6 4 313-318 (6 pages)
killed by a sharp blow to the neck and exsanguination. The carotid arteries were isolated and cut into helical strips of 1.5- to 2-mm width and 8 to 10-mm length and placed in physiological salt Mechanism of Acetylcholine-induced Endothelium-dependent Relaxation in the Rabbit Carotid Ar tery by M3-receptor Activation Yong-Jin Song 1,2 and Seong-Chun Kwon 2 1 Department of Otolaryngology, Head... The present study were designed to characterize the action mechanisms of acetylcholine (ACh)- induced endothelium-dependent relaxation in arteries precontracted with high K (70 mM). For this, we simultaneously measured both muscle tension and cytosolic free Ca2 concentration ([Ca2]i), using fura-2, in endothelium-intact, rabbit carotid arterial strips. In the artery with endothelium, high K increased both [Ca2]i and muscle tension whereas ACh (10μM)... -
Mechanism of Leptin-Induced Potentiation of Catecholamine Secretion Evoked by Cholinergic Stimulation in the Rat Adrenal Medulla
Dong-YoonLim, Deok-HoChoi, Moo-JinKang 대한생리학회-대한약리학회 The Korean Journal of Physiology & Pharmacology 9 Pages
대한생리학회-대한약리학회 The Korean Journal of Physiology & Pharmacology 2004, Vol.8 No.4 9 227-235 (9 pages)
the ObR mRNA or Mechanism of Leptin-Induced Potentiation of Catecholamine Secretion Evoked by Choliner gic Stimulation in the Rat Adrenal Medulla Dong-Yoon Lim, Deok-Ho Choi, and Moo-Jin Kang Department of Pharmacology, College of Medicine, Chosun University, Gwangju 501-759, Korea The aim of the present study was to examine the effect of leptin on CA release from the isolated perfused model... perfused model of the rat adrenal gland, and to establish its mechanism of action. Leptin (1∼100 ng/ml), when perfused into an adrenal vein of the rat adrenal gland for 60 min, enhanced a dose-dependently the secretory responses of CA evoked by ACh (5.32⁓103 M), DMPP (104 M) and McN-A-343 (104 M), although it alone has weak effect on CA secretion. However, it did not affect the CA secretion evoked by excess K (5.6⁓102 M). Leptin alone produced a... -
Presynaptic Mechanism Underlying Regulation of Transmitter Release by G Protein Coupled Receptors
TomoyukiTakahashi, YoshinaoKajikawa, MasahiroKimura, NaotoSaitoh, TetsuhiroTsujimoto 대한생리학회-대한약리학회 The Korean Journal of Physiology & Pharmacology 8 Pages
대한생리학회-대한약리학회 The Korean Journal of Physiology & Pharmacology 2004, Vol.8 No.2 1 69-76 (8 pages)
a common mechanism among various cell systems and nerve terminals. Intra-terminal Coupling Mechanism between GPCRs and VACCs At the cell somata, heterotrimeric G proteins affects their targets either directly via βγ subunits in a membrane- delimited manner (De Waard et al, 1997), or indirectly via second messengers (for review, see Hille, 1994). What is the intracellular mechanism, which... A variety of G protein coupled receptors (GPCRs) are expressed in the presynaptic terminals of central and peripheral synapses and play regulatory roles in transmitter release. The patch-clamp whole-cell recording technique, applied to the calyx of Held presynaptic terminal in brainstem slices of rodents, has made it possible to directly examine intracellular mechanisms underlying the GPCR-mediated presynaptic inhibition. At the calyx of Held, bath-application of agonists for GPCRs such as GABAB... -
The Mechanism of t-Butylhydroperoxide-Induced Apoptosis in IMR- 32 Human Neuroblastoma Cells
Jung-AeKim, YongSooLee, KeunHuh 대한생리학회-대한약리학회 The Korean Journal of Physiology & Pharmacology 10 Pages
대한생리학회-대한약리학회 The Korean Journal of Physiology & Pharmacology 1999, Vol.3 No.1 3 19-28 (10 pages)
of mRNA and protein and showes characteristic features of cell shrinkage， chromatin condensation， and DNA frag mentation (Kerr et al， 1972). However， the exact mechanism of apoptosis induction by ROS in the neuronal cells is not completely understood. In this study， we investigated the mechanism of apoptosis induction by oxidative stress in IMR-32 human neuroblastoma cells as a... to play an important role in the apoptotic cell death. However, the exact mechanism of oxidative stress-induced apoptosis in neuronal cells is not known. In this study, we induced oxidative stress in IMR-32 human neuroblastoma cells with tert- butylhydroperoxide (TBHP), which was confirmed by significantly reduced glutathione content and glutathione reductase activity, and increased glutathione peroxidase activity. TBHP induced decrease in cell viability and increase in DNA fragmentation, a... -
Mechanism of Catecholamine Secretion Evoked by Lithium from the Isolated Perfused Rat Adrenal Gland
임동윤(Dong-Yoon Lim), 김철(Cheol Kim), 오형근(Hyeong-Geun Oh) 대한약리학회 대한약리학잡지 16 Pages
대한약리학회 대한약리학잡지 1996, 제 32권 제 3호 7 357-372 (16 pages)
secretory effect e- voked by Li-Krebs may be mediated through the same mechanism with that by ouabain. Ouabain is also known to release CA from the perfused cat adrenal gland by a calcium-depen- dent exocytotic mechanism (Garcia et al., 1980). The secretory effect of ouabain is not secon- dary to the release of ACh from the cholinergic nerve terminals present in the adrenal gland, but due to... In the present study, it was attempted to investigate the effect of lithium on catecholamine (CA) secretion from the isolated perfused rat adrenal gland and to clarify the mechanism of its action. Replacement of Na+ (118.4 mM) by lithium in the normal Krebs-bicarbonate solution used to perfuse the gland produced gradually an increased response in the spontaneous catecholamine release, which was peaked at 30 ~ 60 min after its perfusion. Li-Krebs solution was perfused into an adrenal vein for 2...