The purpose of this study was to review the limb apraxia. It includes the evaluation and neurophysiological aspects for limb apraxia. Limb apraxia comprised a wide spectrum of higher-order motor disorders that results from acquired brain disease affecting the performance of skilled and/or learned movements with the forelimbs and is a common sequela of left brain damage that consists of a deficit in performing gestures to verbal command or to imitation. There are two forms in limb apraxia; ideational apraxia and ideomotor apraxia. A assessment of limb apraxia typically includes pantomiming and imitation of transitive, intransitive, and meaningless gestures. Limb apraxia has been attributed to damage confined to the cerebral cortex, cortico-cortical connecting pathways, and basal ganglia.